Vitamin A Vergiftung eines 4 jährigen durch Provitamin A reiche Pflanzen zusammen mit Fisch(leber)öl und Betacarotin (=natürlich vorkommendes Provitamin A) supplement.
"Because of his elevated vitamin A level, we carefully reviewed his diet and supplement intake. He was on a predominantly plant-based diet, and we estimated his vitamin A intake to be between 1528 μg and 3304 μg of retinol activity equivalents (RAE) per day, or 31 087–63 507 international units (IU) per day (recommended intake for children at the age of our patient is 400 μg/d RAE). […] The patient’s main sources of vitamin A included 1–2 cups of kale, 2–3 cups of green vegetables, 2–3 cups of fruit and 4 oz of meat per day (nonliver), plus a multivitamin containing vitamin A as β-carotene (28 μg RAE). He had previously been taking cod liver oil supplements (276 μg/day RAE) for more than 1 year, but had stopped many months previously."
Beta-Carotin hat
nichts mit Vitamin A zu tun. Es ist ein Farbpigment. Vitamin A kommt
ausschließlich in tierischen Produkten vor, weshalb die Bezeichnung von Pflanzen als Hauptquellen für Vitamin A vollkommen lächerlich ist.
Sie sind
gar keine Quelle für Vitamin A. Der Junge hat auch ein
Multivitamin eingenommen, das Beta-Carotin enthielt, und Monate vor seiner "Vergiftung"
aufgehört, Fischleberöl zu konsumieren.
Außerdem waren
15 seiner Laborwerte abnormal.
"In 2014, in the southeast of France, a 62-y-old man with a medical history of hypercholesterolemia treated with atorvastatin caught a tuna weighing approximately 75 kg. He fried its liver, which weighed about 1 kg, and shared it with 2 guests. Two hours after the meal, all 3 people developed nausea, hepatalgia, facial flushing, and severe headaches. The patient went to the emergency department the following morning due to persistence of these symptoms. First, he was thought to have scombroid poisoning and was given antihistamine treatment. However, the PCCM, consulted within the following hours,
suggested a diagnosis of hypervitaminosis A due to a large ingestion of fish liver. The patient was given analgesic treatment.
Standard blood test results were unremarkable, and retinol levels were not measured."
Die Hypervitaminose A wurde also nicht diagnostiziert, sondern nur vorgeschlagen, und sein Retinolwert wurde gar nicht GEMESSEN.
"In 2019, the PCCM was consulted by the pediatric emergency department on Reunion Island regarding a 12-y-old girl with major digestive symptoms combined with a severe headache that began a few hours after having shared the liver of a 200 kg marlin (Istiophoridae family) with her family. This patient, with a medical history of asthma and a strabismus, had an evening meal with her parents and her 18-y-old sister during which they shared the liver of a fish cooked by her mother. She woke the following morning with a frontal and periorbital headache with eye pain and phonophobia and a flushed face; she felt nauseated and vomited approximately 10 times.
Her parents also experienced mild headaches with eye pain and flushing. Her older sister only experienced headaches. […] She was treated with metoclopramide, fluids by infusion, and progressive oral feeding. Serum ionogram, blood count, and blood gas analysis were unremarkable. A blood sample collected 18 h postingestion was sent to mainland France to evaluate retinol levels. Results were obtained within 10 d, showing a retinol level of 1458 mg·L-1 (normal 444–945), (or 5.09 mmol·L-1 [normal 1.55–3.30])."
Vier Menschen haben Marlinleber gegessen, von denen nur einer schwere Symptome entwickelt hat, die vollkommen unbegründet dem angeblich "zu hohen" Retinolwert zugeschrieben wurden.
Eine andere Studie sagt folgendes über Marlin: "Eating piscivore marine fish warrants caution. Whereas seafood is the main source of essential selenium (Se) to proper brain function and to developing spermatozoa,
top predators can contain toxic concentrations of mercury (Hg).
Marlin, a billfish consumed by large segments of the Indo-Pacific tropical and subtropical populations and those from the west Atlantic coast,
commonly contains several mg Hg/kg wet weight (ww) that exceed the maximum level of 0.5-1.0 mg/kg ww established by the US Environmental Protection Agency, the US Food and Drug Administration (2017), and Health Canada (2014)."
"Later in 2019, the PCCM was contacted by a 36-y-old woman with a medical history of hypothyroidism treated with levothyroxine. She had eaten approximately 100 g of monkfish and cod liver 4 times over a period of 15 d.
Eight days after the last meal, she developed headaches, nausea, diarrhea, and diffuse arthralgia. This was when she discovered that she was pregnant (3 wk of gestation).
Retinol blood levels at Day 10 after the last meal were normal."
Muss ich dazu noch irgendwas sagen?
"We report a case of acute hypervitaminosis A resulting from accidental ingestion of vitamin A supplement capsules."
Ja, Supplemente sind giftig. Die haben aber
nichts mit echtem Vitamin A zu tun.
Warum nicht? Weil es Blut bei euch nicht zu kaufen gibt?
Genau.
.
